![Suppression of NF-κB Activation by Entamoeba histolytica in Intestinal Epithelial Cells Is Mediated by Heat Shock Protein 27* - Journal of Biological Chemistry Suppression of NF-κB Activation by Entamoeba histolytica in Intestinal Epithelial Cells Is Mediated by Heat Shock Protein 27* - Journal of Biological Chemistry](https://els-jbs-prod-cdn.jbs.elsevierhealth.com/cms/asset/ef267959-3cfc-4088-8b2d-856049b2cc7a/gr1.jpg)
Suppression of NF-κB Activation by Entamoeba histolytica in Intestinal Epithelial Cells Is Mediated by Heat Shock Protein 27* - Journal of Biological Chemistry
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NEMO Prevents RIP Kinase 1-Mediated Epithelial Cell Death and Chronic Intestinal Inflammation by NF-κB-Dependent and -Independent Functions: Immunity
![pIC-and TNF-induced IEC apoptosis is independent of NF-κB signaling,... | Download Scientific Diagram pIC-and TNF-induced IEC apoptosis is independent of NF-κB signaling,... | Download Scientific Diagram](https://www.researchgate.net/profile/Alexis-Broquet/publication/233840443/figure/fig4/AS:601676209733656@1520462195663/pIC-and-TNF-induced-IEC-apoptosis-is-independent-of-NF-kB-signaling-but-NF-kB-signaling.png)
pIC-and TNF-induced IEC apoptosis is independent of NF-κB signaling,... | Download Scientific Diagram
![NF-κB-iNOS-COX2-TNF α inflammatory signaling pathway plays an important role in methotrexate induced small intestinal injury in rats - ScienceDirect NF-κB-iNOS-COX2-TNF α inflammatory signaling pathway plays an important role in methotrexate induced small intestinal injury in rats - ScienceDirect](https://ars.els-cdn.com/content/image/1-s2.0-S0278691518304149-fx1.jpg)
NF-κB-iNOS-COX2-TNF α inflammatory signaling pathway plays an important role in methotrexate induced small intestinal injury in rats - ScienceDirect
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Intranuclear delivery of synthetic nuclear factor-kappa B p65 reduces inflammasomes after surgery - ScienceDirect
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NF-κB Is a Central Regulator of the Intestinal Epithelial Cell Innate Immune Response Induced by Infection with Enteroinvasive Bacteria | The Journal of Immunology
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Nuclear factor κB is activated in macrophages and epithelial cells of inflamed intestinal mucosa - Gastroenterology
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Intestinal Epithelial Cells Exposed to Bacteroides fragilis Enterotoxin Regulate NF-κB Activation and Inflammatory Responses through β-Catenin Expression | Infection and Immunity
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Inhibition of Oxidant-Induced Nuclear Factor-κB Activation and Inhibitory-κBα Degradation and Instability of F-Actin Cytoskeletal Dynamics and Barrier Function by Epidermal Growth Factor: Key Role of Phospholipase-γ Isoform | Journal of Pharmacology and
![Protective effects of osthole, a natural derivative of coumarin, against intestinal ischemia-reperfusion injury in mice Protective effects of osthole, a natural derivative of coumarin, against intestinal ischemia-reperfusion injury in mice](https://www.spandidos-publications.com/article_images/ijmm/31/6/IJMM-31-06-1367-g05.jpg)
Protective effects of osthole, a natural derivative of coumarin, against intestinal ischemia-reperfusion injury in mice
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Intestinal non-canonical NFκB signaling shapes the local and systemic immune response | Nature Communications
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IKK/NF‐κB and STAT3 pathways: central signalling hubs in inflammation‐mediated tumour promotion and metastasis | EMBO reports
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Nuclear Factor-κB Activation Correlates with Better Prognosis and Akt Activation in Human Gastric Cancer | Clinical Cancer Research
![Figure 2. | Persistent Cyclooxygenase-2 Inhibition Downregulates NF-κB, Resulting in Chronic Intestinal Inflammation in the Min/+ Mouse Model of Colon Tumorigenesis | Cancer Research Figure 2. | Persistent Cyclooxygenase-2 Inhibition Downregulates NF-κB, Resulting in Chronic Intestinal Inflammation in the Min/+ Mouse Model of Colon Tumorigenesis | Cancer Research](https://cancerres.aacrjournals.org/content/70/11/4433/F2.large.jpg)
Figure 2. | Persistent Cyclooxygenase-2 Inhibition Downregulates NF-κB, Resulting in Chronic Intestinal Inflammation in the Min/+ Mouse Model of Colon Tumorigenesis | Cancer Research
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Expression of nuclear factor-kappa B and target genes in gastric precancerous lesions and adenocarcinoma: Association with Helicobactor pylori cagA (+) infection
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